SECONDARY RESEARCH BY WINNING THE FIGHT
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ARGENINE CORRECTED METABOLIC DYSFUNCTION IN CHILDREN WITH MITOCHONDRIAL DISEASES
Abstract: One of the substances in the Deanna Protocol Plan is arginine. Its purpose, along with Alpha ketoglutarate, is to deliver energy to the Kreb’s Cycle and, thereby, help the mitochondria function and prevent cells from dying. This piece of literature shows evidence that arginine is effective in correcting malfunctioning mitochondria.
ALS PATIENTS TEST POSITIVE FOR BORRELIA BACTERIA
Abstract: This article proves that ALS is likely caused by the bacteria that belongs to the genus borrelia. Up until now, borreila was only thought to be the cause of Lyme disease, but Dr. Tedone hypothesizes that it also causes ALS. The borrelia bacteria is very hard to detect in the body because, unlike other bacteria, the spirochete bacteria (spiral shaped bacteria) burrows into the soft tissue and is not present in the blood (other than red blood cells). Most tests for borrelia are inefficient at detecting bacteria in soft tissue.
WOMAN DIAGNOSED WITH MUSCULAR SCLEROSIS ACTUALLY HAD BORRELIA BACTERIA (News Article)
Abstract: Mother diagnosed with MS and facing life in a wheelchair is cured – after she discovered her symptoms were due to a TICK BITE
was diagnosed with
MS in 2010 after an
MRI scan revealed
lesions on her brain
Julia Marshall-Wessendorf, 37, was diagnosed with MS in 2010 after an MRI scan revealed lesions on her brain and spine.
Abstract: Article highlights the death of a woman who was diagnosed with ALS, but was later diagnosed with a Borrelia infection. Multiple other cases with similar outcomes are discussed as well.
Abstract: Video 1 and 2 are lectures by Alan MacDonald on the connection between the Borrelia bacteria and multiple sclerosis, live from a London lecture. Video 3 discusses the connection with Alzheimer’s Disease which, like ALS, is likely linked to bacterial infection with Borrelia.
Borrelia Detected in the Brains of Alzheimer’s Patients
Abstract: The connection between Borrelia bacteria and Alzheimer’s Disease are discussed. Alzheimer’s disease is a neurospirochetosis. Article analyzes the evidence following Koch’s and Hill’s criteria.
Blood Brain Barrier is impaired in ALS patients
Abstract: This study shows that the BBB in someone with ALS does not function normally and certain substances that normally should not be able to pass through the BBB are able to pass through in those with ALS. This is significant because some substances in the Deanna Protocol cannot pass the BBB in healthy patients. However, in those with ALS, who have a malfunctioning BBB, the substances are able to pass through. This allows these substances to make a positive impact on patients with ALS. For the layperson: the blood brain barrier is a highly selective semipermeable border that separates the blood from the brain (BBB). The BBB “chooses” what substances are allowed to cross through to the brain and which are not. Many medications and substances introduced into the blood cannot pass the BBB, which means they cannot impact the brain and nervous system.
ALS from Mercury in Amalgam Fillings
Abstract: A toxin, Mercury, could possibly be a cause of ALS, likely if the borrelia bacteria is already present in the body. The article supports Dr. Tedone’s point that toxins likely play a role in ALS and neurodegenerative disease. In 2009, when Dr. Tedone began his research, mainstream medicine did not accept that toxins play a role in ALS and the popular opinion was that non-genetic forms of ALS happened completely at random. Now, the role of toxins in ALS is documented and becoming more accepted in mainstream medicine. New research has given light to a new theory. According to Dr. Tedone and some others studying neurodegenerative diseases, borrelia bacteria is likely the real cause of neurodegenerative disease. Toxins are likely just an aggravator. What does this mean? Someone can have this bacteria in the body for a long time (even decades) without being sick because the immune system can keep the bacteria under control and prevent it from multiplying enough to cause disease. Dr. Tedone and his colleagues hypothesize that environmental factors (such as toxins and stress) can suppress the immune system and prevent it from keeping the bacteria under control. A suppressed immune system allows the bacteria that’s already there to multiply, attack the nervous system, and cause disease. While the studies below don’t address borrelia bacteria and its role in ALS, they do address the fact that toxins in the environment are strongly correlated with onset of neurodegenerative disease symptoms.
Three Teachers in Same Classroom Suffer from ALS
Abstract: ALS is only present in roughly .00595% of the American population (about 16,000 people out of the 270 million living in the U.S.). Due to the rarity of this disease odds of three unrelated teachers who worked in the same classroom all randomly contracting ALS by chance are extremely low. This leads to the probability that their ALS as brought on by an environmental cause. The full article is only available by paid subscription to medical research journals, but the abstract is here.
Toxins in Blue Green Algae Can Cause Nerve Degeneration
Abstract: This study proves that environmental factors such as toxins (and a particular toxin called BAMA in blue green algae) can cause neurodegenerative disease.
Workplace Exposure to Toxins and ALS
Abstract: This document proves that construction workers and precision metal workers, who are exposed to a variety of toxins in their daily work, are more likely to have ALS.
The Evolving Genetic Risk for Sporadic ALS
Abstract: There is a genetic component to sporadic ALS. Dr. Tedone believes it’s possible that the borrelia bacteria in ALS (when they reproduce) enter the cells and interrupt the energy cycle of the cell. In doing so, they cause gene mutations in the cell.